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Glucocorticoids are potent anti-inflammatories, regardless of the inflammation's cause; their primary anti-inflammatory mechanism is lipocortin-1 (annexin-1) synthesis. Lipocortin-1 both suppresses phospholipase A2 , thereby blocking eicosanoid production, and inhibits various leukocyte inflammatory events ( epithelial adhesion , emigration , chemotaxis , phagocytosis , respiratory burst , etc.). In other words, glucocorticoids not only suppress immune response, but also inhibit the two main products of inflammation, prostaglandins and leukotrienes . They inhibit prostaglandin synthesis at the level of phospholipase A2 as well as at the level of cyclooxygenase /PGE isomerase (COX-1 and COX-2),  the latter effect being much like that of NSAIDs , potentiating the anti-inflammatory effect.
In those children with allergy causing the eczema, they will usually get rashes or more extensive eczema soon after eating a particular food or drinking milk (in the case of babies). If you notice this, remove that food from your child's diet ( if you have a baby, this will need to be done under supervision with an appropriate other formula ). If there is a marked improvement on removal of the suspected food, then the food may have been the cause - cautious reintroduction of the food after 3-4 weeks will enable you to be sure. If on reintroduction of the food, the skin remains clear, the food can continue in the diet. If the skin flares up again, that food needs to be removed from the diet. In those toddlers who do have an allergy to a food causing eczema, a trial of reintroduction can be made in 6 - 12 months time as many children will grow out of these allergies.